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肾病材料翻译样例

发布者:鑫达医学翻译 发布时间:2013-05-20 8:50:52 阅读:
原文:
Studies of the natural history of membranous nephropathy show that once excretory renal function starts to decline, continued deterioration can be expected, which suggests that the really important clinical question in membranous nephropathy is whether treatments that are effective in less severely affected patients are also beneficial in patients showing definite signs of decline in renal function. Thus, findings from prospective RCTs are essential to inform decisions about treatment of this disorder.
 
Idiopathic membranous nephropathy is often managed with immunosuppressive drugs. Until recently, evidence that this nephropathy is autoimmune in origin was circumstantial. However, autoantibodies to the phospholipase A2 receptor (PLA2R1) have now been noted in most affected individuals, and a predisposition to the disorder has been very strongly linked to two genetic regions (one in the MHC and the other in the PLA2R1 gene itself)—findings that support an immunological pathogenesis and provide a rationale for immunosuppressive therapy, especially treatment targeted at B lymphocytes.
 
译文:
研究显示,膜性肾病患者一旦肾功能开始降低,就可能出现持续恶化。这表明,膜性肾病治疗中的关键问题在于:这些针对病情较轻的治疗方案是否可有效治疗肾功能明显下降迹象的患者。因此,前瞻性RCT研究所得结果有助于给出针对该疾病治疗的信息。
 
通常,采用免疫抑制药物治疗特发性膜性肾病。目前,尚无确切依据证实该种肾病源于自身免疫异常,但在多数特发性膜性肾病患者体内检测到磷脂酶A2受体(PLA2R1)的自身抗体。此外,两处遗传学改变,分别发生于主要组织相容性复合基因(MHC)与PLA2R1基因,与患者发病高度相关。这些证实,特发性肾病发病可能源于免疫学病变,进而为该疾病的免疫抑制疗法(尤其是针对B淋巴细胞的方案)提供理论基础。

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